What is Lyme Disease

Pathogenic Cause

Lyme disease is a Western medical diagnosis made by a positive antibody blood test of the single generation spirochete bacteria, borrelia Bougdorferi. There are over 300 borrelia documented types of spirochetes, as well many named and un-named spirochetes; all of which, their DNA is always in a state of flux. This makes antibody and PCR testing very difficult to nail down exact infections. Thousands, if not millions of spirochetes can be responsible for similar or same symptoms associated with the single spirochete pathogen associated with the positive borrelia Bougdorferi test.

Lyme disease is taken after the syphilis model. The syphilis spirochete, Treponema pallidum and the Lyme disease spirochete, borrelia Bougdorferi are very similar. They have the same appearance; they behave the same way in the body, and they cause the same type of damage. Differentiating factors are not relevant in this case.1

Though there are many different species and phylum of spirochetes. They look the same, behave the same, and cause the same type of destruction in their host. Many of them are microscopic and some can even be seen with the naked eye. The move in a corkscrew manner, burrowing through the body between tissue types, with the ability to get anywhere and everywhere in the body.

Spirochetes Have Been Found Every Tissue in the Body

Spirochetes are considered a microscopic bacteria that reproduces asexually. They root through the body in a corkscrew manner through the tissues of the body. They can develop cystic and biofilm formations that help them hide from the immune system as well antibiotics. They also mutate their DNA to where their DNA is close to the DNA of their host, which causes the immune system to target its own tissues. They allow the bloodstream to carry them to different parts of the body to infect and degenerate other parts of the body. They leave a trail of toxic waste that cause neurological symptoms in their wake as they move through the body. Spirochetes can live inside a cyst and continue to multiply for up to 8 months without feeding on the body. 

Spirochetes are found in the blood. They can be found in the plasma, in the red blood cells and in the vascular tissue. Spirochetes feed on cartilage, nerve sheath tissue, ligamentous tissue, tendon, bone, organ tissue, fascia, as well any tissue in the body. They contribute to slow or rapid degeneration of the body based on depending factors such as the strength of the immune system, genetics, age, circulation and more. A pathogenic cause should always be considered with any type of degenerative disease presentation, but rarely is.

The first type of spirochete to be researched was the syphilis spirochete. Dr. Hidayo Naguchi researched the syphilis spirochete from 1913-1920. He confirmed the presence of syphilis in brain tissues of cadavers deceased with general paresis. Dr. John Hinchman Stokes MD, PhD (1885-1961) wrote the text Modern Clinical Syphilology, otherwise known as The Green Monster, 1944. The Modern Clinical Syphilology discusses findings of syphilis (Treponemas pallidum) everywhere in the body; such as manifestations in the skin, eyes, nose, nerves, genitals, bones, muscles, fascia, ligaments, tendons and the brain; particularly in the brains of cadavers who died with Alzheimer’s or dementia with general paresis. Microbiologist and researcher of Lyme borrelia pathogens, Dr. Alan McDonald, MD, PhD discovered syphilis with general paresis is a carbon copy of Alzheimer’s disease. Dr. Alan McDonald made this discovery by researching brain tissue samples of Alzheimer’s disease cadavers from Harvard Medical School. Dr. Alan McDonald found spirochetes inside the beta amyloid plaques, associated with Alzheimer’s disease in the human brain tissue. He specifically stated, “I about fell off my chair when I saw spirochetes morph with human brain tissue. Their DNA was 1⁄2 spirochete, 1⁄2 human brain tissue.” This was a new medical discovery at that time. The United States Center of Disease Control and would not publish Dr. Alan McDonald’s findings in fear it would cause public concern. Dr. Alan McDonald went on to state, if his dream comes true some-day, his findings will redefine how medicine is practiced, as well redefine the lives of millions of people.

The Invitation of Other Pathogens

When the immune system is compromised with Lyme disease borrelia, other pathogenic microbes have an easier time invading the body and proliferating at a more rapid rate. Symptoms of Lyme disease is rarely just associated with the Lyme disease borrelia Bougdorferi pathogen, but instead many different pathogens. A common few of billions of other pathogens may consist of co-infections such as bartonella, babesia, anaplasma and erlichia. Others may be mold, systemic candida, and parasites. Others may be bacteria from what we were exposed to in utero or other previous circumstances, such as strep, staphylococcus, tuberculosis, mycoplasma and more. Others may be sexually transmitted diseases such as gonorrhea, syphilis, HPV, herpes simplex virus and more. In a sense, people become carriers of different unwanted microbes. When the immune system becomes compromised, these unwanted microbes come out of hiding, feed on the body, and proliferate at a more rapid rate. The immune system then senses them coming out of hiding, reacts and causes immune reactions such as fevers, chills, nausea and more.

Symptom Development

Chronic symptom clusters are generally caused by pathogenic influence. The Lyme disease borrelia spirochete is one of a gazillion pathogens that can trigger the immune system to cause systemic chronic immune reactions such as but not limited to headaches (inflammation), head pressure, runny nose, post nasal drip, sneezing fits, inflammation of any tissue anywhere of the body (joint paint, flank pain, bloating, abdominal pain, eye pain, difficulty swallowing, ear pain, angina, difficulty breathing, shortness of breath, brain fog, short term memory loss, slow urinary flow, muscular pain, glute pain, neck pain, back of the head pain, lower back pain, spine stiffness and pain, bladder pain, reproductive inflammation and pain, anxiety, depression, and the list goes on and on and on, in regards to symptoms that inflammation can cause).

Disease Development

Though the Lyme disease borrelia pathogen is common to present with inflammation, fibromyalgia, joint pain, brain fog/short term memory loss and fatigue, limitless neurological disease manifestations can also present. After inflammation has been constant and symptoms have been chronic for some time, disease manifestations can develop. There is infinite potential to what disease manifestations can present with. Some may be early onset Alzheimer’s disease, vision loss, hearing loss, stomach ulcers, IBS, ulcerative colitis, Crohn’s, liver disease (fibrosis, cirrhosis), bladder disease, interstitial cystitis, cardiovascular disease of many kinds, reproductive disorders, thyroid disease, polyneuropathy, seizure disorders, MS, ALS, and many more.

To be Continued with Further Posts

Diagnoses

• Western Medical Considerations and Diagnoses and Treatments
• Chinese Medicine Diagnoses and Treatments
• Blending Western and Eastern Medicine Outlook Treatments